Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation
Identifieur interne : 000463 ( Main/Corpus ); précédent : 000462; suivant : 000464Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation
Auteurs : James J. C. Ong ; Jolene M. Kriett ; Gregory K. Feld ; Peng-Sheng ChenSource :
- Journal of Cardiovascular Electrophysiology [ 1045-3873 ] ; 1997-04.
English descriptors
Abstract
Retrograde Conduction in Atrial Fibrillation. Introduction: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation. Methods and Results: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec. Conclusions: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. (2) The presence of an excitable gap in human atrial fibrillation was suggested by atrial preexcitation during retrograde conduction.
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DOI: 10.1111/j.1540-8167.1997.tb00803.x
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Feld</name><affiliation><mods:affiliation>Division of Cardiology, Department of Medicine, UCSD Medical Center, San Diego, California</mods:affiliation></affiliation></author><author><name sortKey="Chen, Peng Heng" sort="Chen, Peng Heng" uniqKey="Chen P" first="Peng-Sheng" last="Chen">Peng-Sheng Chen</name><affiliation><mods:affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:A2CBEB0BED5B611A2E6E316A4FE252E874D7841C</idno><date when="1997" year="1997">1997</date><idno type="doi">10.1111/j.1540-8167.1997.tb00803.x</idno><idno type="url">https://api.istex.fr/document/A2CBEB0BED5B611A2E6E316A4FE252E874D7841C/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">000463</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation</title><author><name sortKey="Ong, James J C" sort="Ong, James J C" uniqKey="Ong J" first="James J. C." last="Ong">James J. C. Ong</name><affiliation><mods:affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</mods:affiliation></affiliation></author><author><name sortKey="Kriett, Jolene M" sort="Kriett, Jolene M" uniqKey="Kriett J" first="Jolene M." last="Kriett">Jolene M. Kriett</name><affiliation><mods:affiliation>Division of Cardiohoracic Surgery, Department of Surgery</mods:affiliation></affiliation></author><author><name sortKey="Feld, Gregory K" sort="Feld, Gregory K" uniqKey="Feld G" first="Gregory K." last="Feld">Gregory K. Feld</name><affiliation><mods:affiliation>Division of Cardiology, Department of Medicine, UCSD Medical Center, San Diego, California</mods:affiliation></affiliation></author><author><name sortKey="Chen, Peng Heng" sort="Chen, Peng Heng" uniqKey="Chen P" first="Peng-Sheng" last="Chen">Peng-Sheng Chen</name><affiliation><mods:affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Journal of Cardiovascular Electrophysiology</title><idno type="ISSN">1045-3873</idno><idno type="eISSN">1540-8167</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1997-04">1997-04</date><biblScope unit="volume">8</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="377">377</biblScope><biblScope unit="page" to="387">387</biblScope></imprint><idno type="ISSN">1045-3873</idno></series><idno type="istex">A2CBEB0BED5B611A2E6E316A4FE252E874D7841C</idno><idno type="DOI">10.1111/j.1540-8167.1997.tb00803.x</idno><idno type="ArticleID">JCE377</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1045-3873</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Wolff‐Parkinson‐White syndrome</term><term>excitable gap</term><term>retrograde conduction</term><term>wavefronts</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Retrograde Conduction in Atrial Fibrillation. Introduction: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation. Methods and Results: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec. Conclusions: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. (2) The presence of an excitable gap in human atrial fibrillation was suggested by atrial preexcitation during retrograde conduction.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>JAMES J.C. ONG M.D.</name><affiliations><json:string>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</json:string></affiliations></json:item><json:item><name>JOLENE M. KRIETT M.D.</name><affiliations><json:string>Division of Cardiohoracic Surgery, Department of Surgery</json:string></affiliations></json:item><json:item><name>GREGORY K. FELD M.D.</name><affiliations><json:string>Division of Cardiology, Department of Medicine, UCSD Medical Center, San Diego, California</json:string></affiliations></json:item><json:item><name>PENG‐SHENG CHEN M.D.</name><affiliations><json:string>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Wolff‐Parkinson‐White syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>excitable gap</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>wavefronts</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>retrograde conduction</value></json:item></subject><articleId><json:string>JCE377</json:string></articleId><language><json:string>eng</json:string></language><abstract>Retrograde Conduction in Atrial Fibrillation. Introduction: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation. Methods and Results: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec. Conclusions: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. 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Ong, M.D., Section of Electrophysiology, Division of Cardiology, Cedars‐Sinai Medical Center, Room 5342, 8700 Beverly Boulevard, Los Angeles, CA 90048, Fax:310‐289‐0780.</p></note><affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</affiliation></author><author><persName><forename type="first">JOLENE M.</forename><surname>KRIETT</surname></persName><roleName type="degree">M.D.</roleName><affiliation>Division of Cardiohoracic Surgery, Department of Surgery</affiliation></author><author><persName><forename type="first">GREGORY K.</forename><surname>FELD</surname></persName><roleName type="degree">M.D.</roleName><affiliation>Division of Cardiology, Department of Medicine, UCSD Medical Center, San Diego, California</affiliation></author><author><persName><forename type="first">PENG‐SHENG</forename><surname>CHEN</surname></persName><roleName type="degree">M.D.</roleName><affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</affiliation></author></analytic><monogr><title level="j">Journal of Cardiovascular Electrophysiology</title><idno type="pISSN">1045-3873</idno><idno type="eISSN">1540-8167</idno><idno type="DOI">10.1111/(ISSN)1540-8167</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1997-04"></date><biblScope unit="volume">8</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="377">377</biblScope><biblScope unit="page" to="387">387</biblScope></imprint></monogr><idno type="istex">A2CBEB0BED5B611A2E6E316A4FE252E874D7841C</idno><idno type="DOI">10.1111/j.1540-8167.1997.tb00803.x</idno><idno type="ArticleID">JCE377</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1997</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Retrograde Conduction in Atrial Fibrillation. Introduction: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation. Methods and Results: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec. Conclusions: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. (2) The presence of an excitable gap in human atrial fibrillation was suggested by atrial preexcitation during retrograde conduction.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>Wolff‐Parkinson‐White syndrome</term></item><item><term>excitable gap</term></item><item><term>wavefronts</term></item><item><term>retrograde conduction</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1997-04">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/A2CBEB0BED5B611A2E6E316A4FE252E874D7841C/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8167</doi><issn type="print">1045-3873</issn><issn type="electronic">1540-8167</issn><idGroup><id type="product" value="JCE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY">Journal of Cardiovascular Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="04004"><doi origin="wiley">10.1111/jce.1997.8.issue-4</doi><numberingGroup><numbering type="journalVolume" number="8">8</numbering><numbering type="journalIssue" number="4">4</numbering></numberingGroup><coverDate startDate="1997-04">April 1997</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0037700" status="forIssue"><doi origin="wiley">10.1111/j.1540-8167.1997.tb00803.x</doi><idGroup><id type="unit" value="JCE377"></id></idGroup><countGroup><count type="pageTotal" number="11"></count></countGroup><titleGroup><title type="tocHeading1">ORIGINAL ARTICLES</title></titleGroup><eventGroup><event type="firstOnline" date="2007-04-20"></event><event type="publishedOnlineFinalForm" date="2007-04-20"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-10"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-29"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-23"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="377">377</numbering><numbering type="pageLast" number="387">387</numbering></numberingGroup><correspondenceTo>Address for correspondence: James J.C. Ong, M.D., Section of Electrophysiology, Division of Cardiology, Cedars‐Sinai Medical Center, Room 5342, 8700 Beverly Boulevard, Los Angeles, CA 90048, Fax:310‐289‐0780.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:JCE.JCE377.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Manuscript received 17 May 1996; Accepted for publication 11 January 1997.</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="20"></count><count type="linksCrossRef" number="5"></count></countGroup><titleGroup><title type="main">Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>JAMES J.C.</givenNames><familyName>ONG</familyName><degrees>M.D.</degrees></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>JOLENE M.</givenNames><familyName>KRIETT</familyName><degrees>M.D.</degrees></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a3"><personName><givenNames>GREGORY K.</givenNames><familyName>FELD</familyName><degrees>M.D.</degrees></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>PENG‐SHENG</givenNames><familyName>CHEN</familyName><degrees>M.D.</degrees></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="US"><unparsedAffiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</unparsedAffiliation></affiliation><affiliation xml:id="a2"><unparsedAffiliation>Division of Cardiohoracic Surgery, Department of Surgery</unparsedAffiliation></affiliation><affiliation xml:id="a3" countryCode="US"><unparsedAffiliation>Division of Cardiology, Department of Medicine, UCSD Medical Center, San Diego, California</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Wolff‐Parkinson‐White syndrome</keyword><keyword xml:id="k2">excitable gap</keyword><keyword xml:id="k3">wavefronts</keyword><keyword xml:id="k4">retrograde conduction</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Retrograde Conduction in Atrial Fibrillation. <i>Introduction</i>: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation.</p><p> <i>Methods and Results</i>: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec.</p><p> <i>Conclusions</i>: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. (2) The presence of an excitable gap in human atrial fibrillation was suggested by atrial preexcitation during retrograde conduction.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="n-fnt-1" numbered="no"><p>This work was done during the tenure of an ACC/Merck Fellowship and a National Institutes of Health NRSA (HL0930201) to Dr. Ong, and an AHA/Wyeth‐Ayerst Established Investigatorship Award (93002670) to Dr. Chen, and was supported in part by a National Institutes of Health FIRST Award (HL50259), a National Institutes of Health SCOR for sudden death (HL52319), and an American Heart Association National Center Grant‐in‐Aid (92009820).</p></note><note xml:id="n-fnt-2" numbered="no"><p>Part of this paper was presented as an abstract at the 45th Annual Scientific Session of the American College of Cardiology, Orlando, Florida, March 25‐27, 1996.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Prevalence of Retrograde Accessory Pathway Conduction During Atrial Fibrillation</title></titleInfo><name type="personal"><namePart type="given">JAMES J.C.</namePart><namePart type="family">ONG</namePart><namePart type="termsOfAddress">M.D.</namePart><affiliation>Division of Cardiology, Department of Medicine, Cedars‐Sinai Medical Center and the School of Medicine, UCLA, Los Angeles, California</affiliation><description>Correspondence: Address for correspondence: James J.C. 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Introduction: Although atrial fibrillation occurs frequently in patients with the preexcitation syndrome, its pathogenesis remains controversial. The purpose of this study was to test the hypothesis that retrograde conduction over the accessory pathway occurs during atrial fibrillation and can serve as an important source of new wavefronts in atrial fibrillation. Methods and Results: Eight patients undergoing surgical division of their accessory path‐way(s) were studied. A plaque electrode array containing 56 (7 × 8) bipolar electrodes (5‐mm resolution) was placed epicardially at the AV junction over the accessory pathway and atrial fibrillation was electrically induced. Excluding one patient who had only preexcited QRS complexes during atrial fibrillation and another whose accessory pathway was outside the mapped region, 4 of the 6 patients studied showed retrograde conduction over the accessory pathway during atrial fibrillation (mean atrial cycle length 157 ± 59 msec). In these patients, 186 atrial wavefronts near the accessory pathway were analyzed. Among 67 wavefronts immediately following nonpreexcited QRS complexes, 17 originated from retrograde conduction. This constituted 9% (17/186) of total atrial wavefronts near the accessory pathway. Estimated atrial refractory period during atrial fibrillation ranged from 81 to 165 msec. Conclusions: (1) In patients with the preexcitation syndrome and atrial fibrillation, retrograde conduction over the accessory pathway contributed up to 9% of total atrial wavefronts near the accessory pathway. (2) The presence of an excitable gap in human atrial fibrillation was suggested by atrial preexcitation during retrograde conduction.</abstract><subject lang="en"><genre>Keywords</genre><topic>Wolff‐Parkinson‐White syndrome</topic><topic>excitable gap</topic><topic>wavefronts</topic><topic>retrograde conduction</topic></subject><relatedItem type="host"><titleInfo><title>Journal of Cardiovascular Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">1045-3873</identifier><identifier type="eISSN">1540-8167</identifier><identifier type="DOI">10.1111/(ISSN)1540-8167</identifier><identifier type="PublisherID">JCE</identifier><part><date>1997</date><detail type="volume"><caption>vol.</caption><number>8</number></detail><detail type="issue"><caption>no.</caption><number>4</number></detail><extent unit="pages"><start>377</start><end>387</end><total>11</total></extent></part></relatedItem><identifier type="istex">A2CBEB0BED5B611A2E6E316A4FE252E874D7841C</identifier><identifier type="DOI">10.1111/j.1540-8167.1997.tb00803.x</identifier><identifier type="ArticleID">JCE377</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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